Myocardial ischemia Treatment in Hyderabad

Overview

Illustration shows the causes of myocardial ischemia
Causes of Myocardial Ischemia Open pop-up dialog
Myocardial ischemia survives when the blood flow to your heart is reduced, which wants the kardiac muscle to receive sufficient oxygen. The decrease in flow is the result of the blockage you received in the Coronary Arteries (Coronary Arteries).

Myocardial ischemia, also called cardiac ischemia, reduces the ability of the heart muscle to pump blood. A sudden and severe blockage of any of the arteries in the heart can lead to a heart attack. Myocardial ischemia can also cause serious heart rhythm abnormalities.

Treatment of myocardial ischemia consists of a flow directed against the heart muscle. Treatment may include medication, a procedure to open blocked arteries (angioplasty), or bypass surgery.

Making healthy lifestyle choices for the heart is the most important treatment and prevention of myocardial ischemia.

symptoms

Some people with myocardial ischemia do not have any signs or symptoms (silent ischemia).

When They Occur and Common is Pressure or Chest Pain, General Assembly on the Left Side of the Body. Other signs and symptoms – which might be experienced more frequently by women, the elderly and people with diabetes

The causes

Development of atherosclerosis
Development of Atherosclerosis Open Contextual Dialog
Myocardial ischemia survives when blood flow to one or more of your coronary arteries is reduced. Poor blood flow reduces the amount of oxygen your heart muscle receives.

Myocardial ischemia can develop slowly as the arteries become blocked over time. Or it can happen quickly when an artery suddenly becomes blocked.

Conditions that can pollute myocardial ischemia include:

Risk factors

Factors that may increase your risk of developing myocardial ischemia include:

Prevention

The same lifestyle habits that can help lock down myocardial ischemia can also help prevent it from developing in the first place. Leading a heart-healthy lifestyle .

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